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patients with alcoholic liver disease only one trial with 123 patients used adequate methodology and reported clearly on mortality and liver transplantation rates. Hardy et al. (2002) noted that few studies had looked at the responses to different oral doses of SAMe and so there was little information available to indicate a likely effective dose of SAMe. Dosages recommended by manufacturers of the product vary between 200 and 1600 mg/day. Hardy et al. (2002) found no evidence from their review of clinical trials that there are any signi cant adverse effects associated with the use of SAMe. There has been speculation that SAMe might raise blood levels of homocysteine which is an independent risk factor for heart disease. However one pharmacological study found that oral SAMe lowered blood levels of homocysteine and had no adverse effects on homocysteine metabolism. Several other claims have been made for bene cial effects of SAMe particularly for bromyalgia ( brositis) which is an arthritis-like condition which affects muscles and tendons rather than joints. Hardy et al. (2002) suggested that the data available for other conditions would almost certainly be less advanced than that for the conditions that they reviewed, depression, osteoarthritis and liver disease.

public void testFindCustomerByName() throws Exception { CustomerPO customer = new CustomerPO(); customer.setFirstName( Al ); customer.setLastName( Capone ); // print out the presentation object System.out.println( Al Capone = + customer); InitialContext ic = new InitialContext();

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P1: JYS WY022-18 WY022/Pfaffenberger WY022/Pfaffenberger-v3.cls June 8, 2004 14:58

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12. The eigenvalues are 5 and 1. Eigenvectors are [1 1]T and [1 A general solution is 1 1 y c1 [ ] e 5t c2 [ ] et. 1 1 From this and the initial conditions we obtain c1 c2 0. This gives the answer y1 7e 5t, y2 c2 7e 5t. 7, c1

(3.4)

model mimics the structure of a rifted continental margin (Section 77) where crustal thickness decreases linearly from right to left and high lithospheric temperatures occur at shallow depths below the thinnest crust In all three models, multiple faults form in the brittle upper crust during the rst 1 2 Ma The number of active faults gradually decreases over time until a single fault dominates the upper crust at about 2 Ma Over time a zone of high strain rate in the ductile lower crust and mantle lithosphere narrows and stabilizes These results show that, for each model, strain localizes where lithospheric strength is at a minimum, regardless of the cause of the weakening They also show that crustal thickness and the initial thermal state of the lithosphere play key roles in localizing strike-slip deformation.

5

These effects may explain why strike-slip faulting has localized in some areas of the southwestern USA (Figs 79, 81), such as the Eastern California Shear Zone and Walker Lane, leaving others, such as the Great Valley Sierra Nevada and central Great Basin, virtually undeformed (Bennett et al, 2003) In this case, strain localization may be related to differences in heat ow between the western Basin and Range Province and the Sierra Nevada (Section 73) However, it has not been demonstrated whether the elevated heat ux is a cause or a product of strain localization Alternatively, crustal thickness variations and horizontal gradients in gravitational potential energy and viscosity may concentrate the deformation (Section 763) In addition to horizontal variations in strength, a vertical strati cation of the lithosphere into weak and strong layers greatly in uences how strain is accommodated during strike-slip deformation.

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